Download Beneficial Effects of Fish Oil on Human Brain by Akhlaq A. Farooqui PDF
By Akhlaq A. Farooqui
It is turning into more and more obtrusive that the deficiency of n-3 fatty acids in vitamin isn't just linked to cardiovascular illnesses, but in addition thinking about stroke, epilepsy and different neurological and neurodegenerative ailments, akin to Alzheimer affliction, Parkinson affliction, and peroxisomal ailments. In his most recent e-book, Beneficial results of Fish Oil on Human Brain, Dr. Akhlaq A. Farooqui expands at the prestige and healing value of n-3 fatty acids (major parts of fish oil) and their mediators in common mind and people with neurodegenerative and neuropsychiatric illnesses. Farooqui offers some great benefits of n-3 fatty acids on Western vitamin, that is enriched in n-6 fatty acids (major parts of vegetable oil) and should advertise the above neurological problems. The booklet will current readers with innovative and accomplished info on metabolism and roles of neural membrane n-3 fatty acids.
Neuroscientists, clinicians, neurologists, neuropathologists, opthamologists, dietitians, and nutritionists will locate this ebook priceless for realizing the molecular foundation of n-3 fatty acids and their lipid mediators (resolvins and neuroprotectins) in neuroprotection in acute neural trauma (stroke, spinal twine trauma and head harm) and neurodegenerative diseases.
About the Author:
Dr. Akhlaq A. Farooqui is a pace-setter within the box of mind phospholipases A2, bioactive ether lipid metabolism, polyunsaturated fatty acid metabolism, glycerophospholipid-, sphingolipid-, and cholesterol-derived lipid mediators, and glutamate-induced neurotoxicity. He has came across the stimulation of plasmalogen-selective phospholipase A2 (PlsEtn-PLA2) and diacyl- and monoacylglycerol lipases in brains from sufferers with Alzheimer affliction. Stimulation of PlsEtn-PLA2 produces plasmalogen deficiency and raises degrees of eicosanoids that could be on the topic of the lack of synapses in brains of sufferers with Alzheimer disorder. Dr. Farooqui has released leading edge learn at the iteration and identity of glycerophospholipid-, sphingolipid-, and cholesterol-derived lipid mediators in kainic acid neurotoxicity via lipidomics. He has formerly authored 4 monographs: Glycerophospholipids in mind: Phospholipase A2 in Neurological Disorders (2007); Neurochemical features of Excitotoxicity (2008); Metabolism and features of Bioactive Ether Lipids in Brain (2008); and Hot issues in Neural Membrane Lipidology (2009). All monographs are released by means of Springer.
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Furthermore, fish oil ingestion also modulates lipid metabolism-related enzyme activity, fatty acid b-oxidation, o-oxidation, and malic enzyme activities in the small intestine of mice fed the 8% fish oil diet compared to mice fed the 30% triacylglycerol-enriched diet. , 2007). , 2008). The upregulated genes in female liver are associated with proteosome endopeptidase activity, lipid metabolism, alcohol metabolism, mitochondrial and oxidoreductase activity. The downregulated genes are associated with oxidoreductase activity, chaperone activity, and electron transport activity in the female liver as shown by biological theme analysis.
1992). Stimulation of PKC and oleic acid-mediated inhibition of gap junction can be prevented by inclusion of bovine serum albumin. The physiological significance of oleic acidmediated gap junction inhibition is not fully understood. However, it is well known that oleic acid increases the rate of cell proliferation not only by enhancing glucose uptake but also by stimulating PKC. Both these factors are critical for neural cell proliferation. , 1997), and activation of PKC is associated with neural cell proliferation.
2005). Chronic hyperglycemia is an additional risk factor for cardiovascular disease. Diabetes patients have two- to four fold increased risk of dying from complication of cardiovascular disease (Colwell, 1997). , 1998; Garg and Grundy, 1990). , 2004). In non-diabetic and diabetic rats, levels of triacylglycerol are decreased by 69% and 20%, respectively, compared with control rats. Fish oil treatment decreases non-esterified fatty acids (NEFA) by 29% in diabetic rats, but the NEFA levels in non-diabetic rats are not affected.