By Ruth Duncan, Helmut Ringsdorf, Ronit Satchi-Fainaro (auth.), Ronit Satchi-Fainaro, Ruth Duncan (eds.)
1 Duncan, H. Ringsdorf, R. Satchi-Fainaro: Polymer Therapeutics: Polymers as medicines, Drug and Protein Conjugates and Gene supply structures: earlier, current and destiny Opportunities.- 2 P.K. Dhal, S.R. Holmes-Farley, C.C. Huval, T.H. Jozefiak: Polymers as Drugs.- three R.J. Amir, D. Shabat: Domino Dendrimers.- four G. Pasut, F.M. Veronese: PEGylation of Proteins as adapted Chemistry for Optimized Bioconjugates.- E. Wagner, J. Kloeckner: Gene supply utilizing Polymer Therapeutics.-
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Additional resources for Polymer Therapeutics I
1 Sequestration of Clostridium Difficile Toxin Clostridium difﬁcile (C. difﬁcile) is responsible for large numbers of episodes of diarrhea that arise from anti-bacterial treatment in nosocomial settings . Since normal colonic ﬂora inhibit the growth of C. difﬁcile, the outbreak of this disease has been attributed to the disruption of normal colonic ﬂora by antibiotic treatment, as is prevalent in hospital settings. C. difﬁcile releases two high molecular weight proteins (toxins A and B), which are the primary causes of diarrhea in patients with C.
Both polymers exhibit lower rates of side effects and are better tolerated than previously marketed BAS. These BAS have been recommended for use as monotherapy or in combination therapy by co-administration with other cholesterol lowering drugs, such as statins. 3 Polyvalent Interactions and Anti-Infective Polymeric Drugs Polyvalent interactions are characterized by the simultaneous binding interaction between multiple ligands on one molecular entity and multiple receptors on another (cells, viruses, proteins, etc).
Cell-surface glycoproteins . This biological process of viral invasion of mammalian host cells is schematically shown in Fig. 9. One of the potential strategies to treat inﬂuenza virus infection is to block the binding of the virus to mammalian cells by presenting polymers bearing several sialic acid groups as competitors for cell surface ligands . Although individual viral surface Fig. 9 Schematic illustration of the biological process involving viral invasion of mammalian host cells Table 4 Representative examples of polyvalent ligands as inﬂuenza virus inhibitors Polymers as Drugs 39 hemagglutinin (HA) binds individual cell-surface sialic acids (SA) weakly, the virus attaches to the cellular surface through multiple interactions between clusters of HA and SA residues.